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Prednisone is used for treating severe allergies, arthritis, asthma, multiple sclerosis, and skin conditions.

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Dosage of oral prednisone is a common regimen for management of advanced prostate cancer. The most widely used oral antiandrogens are finasteride (propranolol) and isotretinoin (Accutane) which have been shown to be effective at reducing the progression of prostate cancer (1); these are used to inhibit the expression of androgenic receptor, and thereby reduce the growth of prostate (2,3). primary benefit these drugs over their less expensive analogs is the relative lack of side effects (2). The effects of these medications on normal, as well cancerous prostate tissue, have been studied previously. Both treatment regimens, finasteride and isotretinoin, are effective for the treatment of prostate cancer as evidenced by dramatic reductions in prostate-specific antigen (PSA), antigens (PSA-PSA), and prostate volume; however, the effects on tumor biology are quite different. Finasteride exhibits a more profound effect on prostate cancer cells as evidenced by its suppression of cell division, decreased numbers cancer cells, and increased apoptosis of cancer cells (1). This is the primary mechanism by which finasteride protects prostate cancer from apoptosis, while isotretinoin is anti-apoptotic and acts primarily against cancer oral prednisone dosage for allergy cells (1,2). In addition to decreased PSA, Finasteride and isotretinoin have been found to inhibit prostate cancer growth in multiple models (1,2). Specifically, both drugs can decrease the amount of prostate tumor cells by 30%, to as much 70%, in vitro (4,5). Finasteride is the most active of two drugs, although it must be combined with progesterone (prostate specific antigen) in order to be optimal (1). In addition, Finasteride has been shown to reduce cancer progression by 25% and increase the median survival time of patients by 14% (1,3); isotretinoin has less effect on the number of tumors but does reduce the progression rates of prostate cancer patients by 13% (1). While these drugs have a profound effect on the tumor tissue, it is ability to reverse prostate tumor cells that poses the greatest challenge for treatment of this disease. Finasteride and isotretinoin both possess potent anti-invasive compounds (5). Prostate cancer cells are capable of spreading and can also divide rapidly, this ability, along with their tendency to undergo apoptosis (cell death), provides ample opportunity for these drugs to reduce the size of tumor (1,5). Several mechanisms by which these drugs suppress tumor growth have been Prednisone 20mg $80.59 - $0.45 Per pill demonstrated. One mechanism involves inhibition of phosphoinositide-3 kinase (P3K). This hormone system, originally found in the brain, regulates cell growth, differentiation, and apoptosis, is especially important for the maintenance of nerve cells prednisone pills online and the control of immune responses. phosphoinositide-3 pathway serves to regulate cell growth, proliferation, differentiation, and survival; inhibition of P3K has been established as a treatment for tumor formation (6). This pathway is of particular interest to the researchers of anticancer drugs, as inhibition the P3K pathway will likely suppress cells that promote the growth of cancer (7). Both medications inhibit P3K activity, and their ability to do so is largely due to the activity of phosphatidylinositol-3 kinase (3,4,6). Another target of the medications is P53 tumor suppressor pathway (4,8). is a protein, first identified in the embryo, that encodes for activated forms of several tumor suppressor genes, a protein that is critical for the repression of tumorigenesis and growth tumors. P53 functions by disrupting the interaction of androgen receptor and its target genes, thereby reducing the expression of these genes.

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